I have discussed this in 2019 in my paper: Neuromuscular dentistry and the role of the autonomic nervous system: Sphenopalatine ganglion blocks and neuromodulation. An International College of Cranio Mandibular Orthopedics (ICCMO) position paper
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Are some of the almost magical effects of Neuromuscular Dentistry related to increased blood flow to the brain from the use of ULF-TENS?
The Sphenopalatine Ganglion sits on the maxillary division of the Trigeminal Nerve in the Pterygoidpalatine fossa, bilaterally.
The Trigeminal nerve controls the blood flow to the meninges of the brain.
ULF-TENS can help correct issues of Trigeminal Nerve origin, ULF-TENS is primarily utilized to relax masticatory muscles in Neuromuscular Dentistry.
The DNO™ or Diagnostic Neuromuscular Orthotic is thought by many top clinicians world-wide to be the best away to treat TMJ Disorders along with many types of headaches including the Autonomic Cephalgias.
This author considers neuromodulation of the Sphenopalatine Ganglion and Nasal blocks of the Sphenopalatine Ganglion to be an essential part of neuromuscular treatment of both TMD and headaches including the Autonomic Cephagias.
Review Clin Auton Res
. 2025 Feb;35(1):31-43. doi: 10.1007/s10286-024-01085-6. Epub 2024 Dec 18.
Sphenopalatine ganglion stimulation for the treatment of cerebrovascular ischemia
Juan Manuel Marquez-Romero 1, Karen Itzel Sánchez-Ramírez 2
Affiliations Expand
PMID: 39692954 DOI: 10.1007/s10286-024-01085-6
Abstract
Purpose: The parasympathetic effects of the sphenopalatine ganglion (SPG) on the cerebral vasculature provide a compelling rationale for its therapeutic application in cerebrovascular ischemia. In recent years, attempts have been made to stimulate the SPG to achieve beneficial effects on cerebral circulation.
Methods: This review synthesizes the available publications on SPG stimulation. It critically evaluates the evidence from preclinical studies and clinical trials to assess its potential as a treatment for acute cerebrovascular disorders and outlines aspects that still require more study.
Results: The medical literature provides consistent evidence of the significant effects of stimulating the SPG on cerebrovascular circulation. In addition, considerable evidence supports the therapeutic role of SPG stimulation in patients with cerebral ischemia.
Conclusion: Given the current understanding, future research could explore the potential of SPG stimulation as a non-reperfusion intervention to improve long-term outcomes for individuals with ischemic cerebrovascular conditions.
Keywords: Chemical; Electric stimulation; Ganglia; Parasympathetic nervous system; Pterygopalatine; Stimulation; Stroke.